Tissue oedema

Tissue oedema

During systemic inflammation, fluid, plasma proteins, leukocytes, macrophages and electrolytes leave the vascular space and accumulate in the tissues as oedema. The oedema - can diminish the alveolar di ff usion of oxygen and may also impair renal function. Increased capillary leak is mediated by a wide variety of mediators, including cytokines, prostanoids, - bradykinin and nitric oxide. Cellular hypoxia and dysfunction can occur. Intracellular volume decreases, and this provides part of the volume necessary to replenish intravascular and extravascular extracellular volume. - Tissue oedema

During systemic inflammation, fluid, plasma proteins, leukocytes, macrophages and electrolytes leave the vascular space and accumulate in the tissues as oedema. The oedema - can diminish the alveolar di ff usion of oxygen and may also impair renal function. Increased capillary leak is mediated by a wide variety of mediators, including cytokines, prostanoids, - bradykinin and nitric oxide. Cellular hypoxia and dysfunction can occur. Intracellular volume decreases, and this provides part of the volume necessary to replenish intravascular and extravascular extracellular volume. - Tissue oedema

During systemic inflammation, fluid, plasma proteins, leukocytes, macrophages and electrolytes leave the vascular space and accumulate in the tissues as oedema. The oedema - can diminish the alveolar di ff usion of oxygen and may also impair renal function. Increased capillary leak is mediated by a wide variety of mediators, including cytokines, prostanoids, - bradykinin and nitric oxide. Cellular hypoxia and dysfunction can occur. Intracellular volume decreases, and this provides part of the volume necessary to replenish intravascular and extravascular extracellular volume. -