Changes to the intestine

Changes to the intestine

The inflammatory stimulus and shock can cause microvascular damage and ischaemia to the gut mucosa. This reduces gut motility and can prevent the absorption of food. Failure of enteral feeding in a patient with a large burn is a life-threatening complication. This process also increases the translocation of gut bacteria, which can become an important source of infection in large burns. Gut mucosal swelling, gastric stasis and peritoneal oedema can also cause abdominal compartment syndrome, which splints the diaphragm and increases the airway pressures needed for respiration. Changes to the intestine

The inflammatory stimulus and shock can cause microvascular damage and ischaemia to the gut mucosa. This reduces gut motility and can prevent the absorption of food. Failure of enteral feeding in a patient with a large burn is a life-threatening complication. This process also increases the translocation of gut bacteria, which can become an important source of infection in large burns. Gut mucosal swelling, gastric stasis and peritoneal oedema can also cause abdominal compartment syndrome, which splints the diaphragm and increases the airway pressures needed for respiration. Changes to the intestine

The inflammatory stimulus and shock can cause microvascular damage and ischaemia to the gut mucosa. This reduces gut motility and can prevent the absorption of food. Failure of enteral feeding in a patient with a large burn is a life-threatening complication. This process also increases the translocation of gut bacteria, which can become an important source of infection in large burns. Gut mucosal swelling, gastric stasis and peritoneal oedema can also cause abdominal compartment syndrome, which splints the diaphragm and increases the airway pressures needed for respiration.