Oral cavity

Oral cavity

All sites in the oral cavity are examined under direct visual - isation. Table 53.4 details the signs and symptoms that are suggestive of a neoplastic process. Figure 53.4 demonstrates the wide clinical presentation of OCSCCs, which range from small areas of (erythro)leukoplakia to larger erosive and cavitated lesions that invade surrounding tissues. Reduced tongue movement, sensory nerve deficit, trismus, otalgia and dysphagia are all in keeping with late-stage disease. Fibreoptic examination is not routinely performed in oral cavity assess - ment but may support assessment of the posterior extent of the tumour. The incidence of synchronous primary tumours of the upper digestive tract is 2.4–4.5%. /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF

TABLE 53.4 Signs and symptoms of oral cavity neoplasm. Signs Symptoms Sensory nerve de /f_i cit Non-healing ulcer (>2 weeks) Chronic otalgia Persistent neck mass/ lymphadenopathy Trismus of unknown aetiology Lesion, pigmentation with progressive increase in size Dysphagia Lesion with associated induration Persistent red or white lesion Non-resolving ‘in /f_l ammatory’ lesion Soft-tissue lesion with associated radiographic changes Unexplained tooth mobility

(c) Figure 53.4 Clinical presentations of oral cavity squamous cell carcinoma (SCC). Note the cardinal features consistent with malignancy; namely speckled appearance, raised rolled edges, contact bleeding and variable ulceration. (c) /uni00A0 Left posterolateral tongue SCC. (d) Right lateral tongue SCC.

Oral cavity

All sites in the oral cavity are examined under direct visual - isation. Table 53.4 details the signs and symptoms that are suggestive of a neoplastic process. Figure 53.4 demonstrates the wide clinical presentation of OCSCCs, which range from small areas of (erythro)leukoplakia to larger erosive and cavitated lesions that invade surrounding tissues. Reduced tongue movement, sensory nerve deficit, trismus, otalgia and dysphagia are all in keeping with late-stage disease. Fibreoptic examination is not routinely performed in oral cavity assess - ment but may support assessment of the posterior extent of the tumour. The incidence of synchronous primary tumours of the upper digestive tract is 2.4–4.5%. /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF /uni25CF

TABLE 53.4 Signs and symptoms of oral cavity neoplasm. Signs Symptoms Sensory nerve de /f_i cit Non-healing ulcer (>2 weeks) Chronic otalgia Persistent neck mass/ lymphadenopathy Trismus of unknown aetiology Lesion, pigmentation with progressive increase in size Dysphagia Lesion with associated induration Persistent red or white lesion Non-resolving ‘in /f_l ammatory’ lesion Soft-tissue lesion with associated radiographic changes Unexplained tooth mobility

(c) Figure 53.4 Clinical presentations of oral cavity squamous cell carcinoma (SCC). Note the cardinal features consistent with malignancy; namely speckled appearance, raised rolled edges, contact bleeding and variable ulceration. (c) /uni00A0 Left posterolateral tongue SCC. (d) Right lateral tongue SCC.